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. 2022 Jun 1;11(1):30. doi: 10.1038/s41389-022-00406-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Chromatin immunoprecipitation followed by QPCR for various MYC-bound loci after ACBI1 treatment for 24 h. E2F2 serves as a negative control for binding (n = 4 biological replicates, error bars are standard error). b Venn diagram comparing all significant transcript changes that result from ACBI1 treatment to decreased (top) or increased (bottom) gene expression changes that occur after removal of MYC using the dTAG approach [15]. c GO analysis of 1137 genes that are commonly decreased in expression across dTAG MYC and ACBI1 samples. Number of genes in each term are shown next to the bar.