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. 2022 Jun 1;11(1):30. doi: 10.1038/s41389-022-00406-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Venn diagram showing genes associated with decreased ATAC-seq peak intensity (fold change < −1.5) and their overlap with genes that are increased or decreased in the RNA-seq analysis. b GO analysis of 809 genes in a that have decreased chromatin accessibility and a subsequent decrease in expression. c Venn diagram showing genes associated with decreased ATAC-seq peak intensity (fold change < −1.5) compared to MYC target genes that had changed gene expression in Fig. 3a. d Venn diagram showing genes associated with decreased ATAC-seq peak intensity (fold change < −1.5) compared to all MYC-bound genes in G401 cells. e Normalized ATAC-peak intensities for DMSO and ACBI1 samples are shown for all MYC-bound genes. ACBI1 peaks are ranked against those in DMSO sample.