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. 2022 Apr 12;13(4):9588–9601. doi: 10.1080/21655979.2022.2054206

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — Luciferase assays showed that miR-21 and miR-30e suppressed the expression of PTEN and miR-21 while inhibiting the expression of lncRNA-CASC7 (* P value < 0.05, vs. control group).

A: Sequence analysis indicated potential binding of miR-21 to lncRNA-CASC7.B: The luciferase activity of wild-type lncRNA-CASC7 was inhibited by miR-21 in TPC-1 cells.C: The luciferase activity of wild-type lncRNA-CASC7 was inhibited by miR-21 in BCPaP cells.D: Sequence analysis indicated potential binding of miR-21 to PTEN.E: The luciferase activity of wild type PTEN was inhibited by miR-21 in TPC-1 cells.F: The luciferase activity of wild type PTEN was inhibited by miR-21 in BCPaP cells.G: Sequence analysis indicated potential binding of miR-30e to PTEN.H: The luciferase activity of wild type PTEN was inhibited by miR-30e in TPC-1 cells.I: The luciferase activity of wild type PTEN was inhibited by miR-30e in BCPaP cells.