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. 2022 May 12;59(7):4368–4383. doi: 10.1007/s12035-022-02856-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — Effects of PIO and TEL with and without PPARγ blocker on neuronal survival in 3-NP rats. Representative cresyl violet-stained striatal sections: (a) control, (b) 3NP group, (c) PIO-pre-treated group, (d) PIO&GW-pre-treated group, (e) TEL pre-treated group, and (f) TEL&GW-pre-treated group. (g) Quantification of intact neurons in striatal sections of the experimental groups. Data are presented as means ± SD (n = 3) and analyzed by one-way ANOVA followed by the Tukey’s post hoc test. @ vs control, # vs 3-NP group, $ vs PIO-pre-treated group, & vs TEL-pre-treated group. (PIO: pioglitazone; TEL: telmisartan; 3-NP: 3-nitropropionic acid; GW: GW9662)