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. 2022 May 16;13:877898. doi: 10.3389/fphar.2022.877898

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Copyright © 2022 Liu, Huang, Wan, Lu, Zhou, Li, Yu, Chou, Zhang, Zhang, Qiang, Zhang and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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UA reduced the seizure susceptibility and inhibited chronic epilepsy development of the model. (A) The latency of seizure occurrence induced by lithium-pilocarpine. (B) The latency of different seizure stage induced by lithium-pilocarpine. (C) Percentages of rats with developed SE induced by pilocarpine. (D–G) Representative traces of EEG activity and spectrograms of representative EEG recordings for 50 min. Bar: 500 μv, 10 min. (H) FFT analysis of EEG recordings. (I) Comparison of the average δ (0.75–4 Hz) power. (J) Diagram of electrode implantation. (K) Number of “EEG seizure” events in each group. Each individual box was color-coded according to the legend. Each of the numbers in these boxes indicated the total number of events per day. (L) Example of events in each group. Bar: 500 μv, 3 s (n = 6, all values are expressed as means ±SD, ^∗∗∗ p < 0.001, ^∗∗ p < 0.01, ^∗ p < 0.05 vs. SE, one-way ANOVA, or two-way ANOVA).