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. 2022 Jun 6;15:192. doi: 10.1186/s13071-022-05291-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Changes in tumor cell ultrastructure after Plasmodium infection. The ultrastructural changes in tumor cells in the two groups were observed by TEM. The damaged cell nuclei and mitochondria were observed in the CT26.WT + P.y group. The mitochondria (M, red arrow) were swollen and the cristae disappeared and vacuolated, with chromatin condensation and nuclear disintegration (N: nucleus). The number of autolysosomes (ASS, blue arrow) in the CT26.WT + P.y group was more than that in the CT26.WT group. a, c The magnification is ×2000, scale bar = 5 µm. b, d The magnification is ×6000, scale bar = 1 µm