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. 2022 Jun 7;20:258. doi: 10.1186/s12967-022-03422-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — miR-18a-5p knockdown partially annulled the inhibition of EVs on OC cell proliferation, migration and invasion. A EVs were treated differently and miR-18a-5p expression in EVs was detected by RT-qPCR; B Size distribution of EVs was analyzed by NTA; C–E miR-18a-5p expression was measured via RT-qPCR; F Cell viability was assessed using MTT assay; G, H Migration and invasion of OC cells were evaluated by Transwell assays. Cell experiments were replicated 3 times. Data were presented as mean ± SD. Data were analyzed by two-way (panel F) or one-way ANOVA (panels A/C/D/E/G/H).One-way ANOVA was employed for comparisons between groups and Tukey's multiple comparisons test was performed for the post hoc test. **P < 0.01, ***P < 0.001