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. 2022 Jun 6;22:470. doi: 10.1186/s12884-022-04791-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Knockdown of SLC35C1 impedes the proliferation and invasion while accelerating the inflammatory response of trophoblast cells in vitro. A The interference efficiency of sh-SLC35C1 was examined. B-C The viability and invasion of trophoblast cells were assessed when SLC35C1 was down-regulated. D-E RT-qPCR and western blot analyzed the expression of IL-6, IL-1β, TNF-α and CXCL-8 after SLC35C1 was silenced. The samples in the last group of western blot experiment were indicated positive controls for corresponding antibodies. ^**P < 0.01