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. 2022 Jun 9;5:565. doi: 10.1038/s42003-022-03521-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Phenotype effect on telomere lengths of different cell types (with at least one significant association). Dashed lines show 95% confidence interval (estimate ± 2xSE). X-scale is symmetrical log-transformed (denominator constant = −2). BMI Body mass index, AAT Alpha-1 Antitrypsin Test. b Total variance of the telomere length explained after removing the effect of age. Only phenotypes associated with at least two cell types (FDR < 0.05) are used. Colour indicates the different partitions of variability. c Mediation effect of methylation of SOX11 in the maternal age effect on NK-cell telomere length variability.