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. 2022 Jun 10;12:9634. doi: 10.1038/s41598-022-13655-7

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© The Author(s) 2022, corrected publication 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Si-based agent alleviated the visceral pain and discomfort associated with UC. Analysis of the neuronal activity influenced by colitis, using immunofluorescence staining with anti-c-Fos antibodies. (A,B) The representative photographs of the dorsal medulla (A) and central amygdaloid nuclei (B) 5 days after 5% DSS treatment. Con Con-DSS group, Si Si-DSS group. The vagal dorsal motor nuclei and solitary tract nuclei (A), and central amygdaloid nuclei (B) of a control mouse (arrow) and an Si-based agent-treated mouse (arrowhead). Square in (B): the central amygdaloid nuclei, 12N: hypoglossal nerve nuclei, AP: area postrema, cc: central canal, and Ce: central amygdaloid nuclei. Scale bar: 200 µm.