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. 2022 May 31;9:898674. doi: 10.3389/fnut.2022.898674

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Copyright © 2022 Miao, Yu, Li, Sun and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The anti-tumor mechanisms of C. militaris-derived polysaccharides. C. militaris-derived polysaccharides induce cancer cell death by enhancing apoptosis-associated signaling. Alternatively, they polarize tumor-associated macrophages from M2 to M1 phenotype, thereby promoting the cancer cell killing activity of T lymphocytes. AKT/PKB, phosphoinositide-3-kinase/protein kinase B; Bax, B cell leukemia/lymphoma 2-associated X protein; Bcl-2, B cell leukemia/lymphoma 2; ERK, extracellular regulated kinase; NF-κB, nuclear factor kappa-B; PD-1, programmed death-1; PD-L1, programmed death lignd-1.