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. 2022 Jun 10;219(8):e20200418. doi: 10.1084/jem.20200418

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© 2022 Zhang et al.

This article is available under a Creative Commons License (Attribution 4.0 International, as described at https://creativecommons.org/licenses/by/4.0/).

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Figure 6. — CCL17 neutralizing antibodies inhibited Ang II–induced inflammatory response. (A–D) Flow cytometry analysis of CD45⁺ T cells (A), CD3⁺CD4⁺ T cells (B), and T cell subsets (C and D) of saline- or Ang II–infused C57BL/6J background WT mice (8–12 wk old) treated with IgG control or anti-CCL17 antibodies (n = 6 biological replicates). (E) Th1/Th2 and Th17/Treg ratios in C57BL/6J background WT mice (8–12 wk old) treated with IgG control or anti-CCL17 antibodies after saline or Ang II infusion (n = 6 biological replicates). All data are presented as the mean ± SD. The Kolmogorov–Smirnov normality test was used to assess data distribution. Statistical significance was tested and obtained using one-way ANOVA, followed by the Bonferroni post hoc test (A–E). **, P < 0.01; ***, P < 0.001.