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. 2022 Jun 14;20:273. doi: 10.1186/s12951-022-01491-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Characteristic and anti-tumor mechanism of the biomimetic nanosystem (DR@PLip). A Preparation and anti-tumor mechanism of DR@PLip. Lipsomes co-loading DOX and Rg3 were co-extruded with PM to construct the DR@PLip. The biomimetic nanosystem in blood encountered the AML cells and induced powerful immunogenic cell death. Then the activated effector T cells penetrated deep in bone marrow to kill the AML cells, as well as eliminated the AML cells remained in blood. B Size distribution and zeta potential of the nanosystem. C TEM images of the nanosystem. Scale bar, 100 nm. D The PLT relative protein detected in different samples