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. 2022 Jun 15;19:150. doi: 10.1186/s12974-022-02483-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Resistance training modulated mitochondria dynamic following surgery. a and b Representative blots and quantitative analysis of mitofusin-1 and mitofusin-2 in the mitochondria fraction, the intensity of band was normalized to that of VDAC. n = 6 for mitofusin-1, 8 for mitofusin-2. c Representative TEM pictures of ER–mitochondria contacts as indicated by the white arrow. d Quantification of ER–mitochondria contacts, demonstrating by the contact area divided by the mitochondrial girth. n = 20 comprised 5 pictures from 4 mice in each group. e–g Representative blots and quantitative analysis of NMDARs and AMPK-α, with the intensity of band normalized to that of β-actin. Data analyzed by two-way ANOVA, followed by Tukey multiple comparisons test, n = 5 (except for Lap group, n = 6 for the expression of proteins in synaptosome). Data presented as mean ± SEM, *p < 0.05, **p < 0.01, ***p < 0.001. M: mitochondria; ER: endoplasmic reticulum