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Mechanisms of QL in ventricular remodeling of the transverse aortic constriction-induced HF model. QL intervention could ameliorate the gut dysbiosis, improve the gut barrier, and inhibit gut-derived inflammatory cytokines entering the circulatory system. The heart-protective effect of QL might associate with the NF-κB relative pathway inhibition and the NLRP3 inflammasome suppression in the cardiac tissue.
