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. 2022 Jun 15;29:42. doi: 10.1186/s12929-022-00824-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — KRT17 silencing potentiates therapeutic efficacies of cisplatin, carboplatin and dasatinib in OSCC. A Dose-dependent cell viability assays were performed to examine the effect of KRT17 silencing on drug sensitivity of C9IV3 and HSC3 cells that were treated with cisplatin or carboplatin ranging from 0 to 100 μM. B Dose-dependent cell viability assays were performed to investigate whether combinatorial treatments of dasatinib with cisplatin or carboplatin could be therapeutically more effective against C9IV3 and HSC3 cells. Data are presented as the mean ± SD (*p < 0.05, **p < 0.01 and ***p < 0.001)