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. 2022 May 23;28(6):1303–1313. doi: 10.1038/s41591-022-01837-9

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 6 — COVID-19 without cardio-renal involvement: no abnormalities identified. A 25-year-old woman presented with breathlessness, lightheadedness and anosmia 9 days after a nasopharyngeal swab tested PCR-positive for SARS-COV-2 infection. The admission electrocardiogram (a) revealed normal sinus rhythm. She was treated with dexamethasone. Research-indicated chest computed tomography (CT) (b, c) and cardio-renal magnetic resonance imaging (MRI) (d, e, f, g) were acquired in line with the protocol 23 days after discharge from hospital. High resolution lung CT revealed faint peribronchovascular ground glass opacification in keeping with recovering pneumonitis. The coronary CT angiogram was normal (C). Renal MRI imaging revealed normal T1 values (D: cortex 1481 ms, medulla 1922ms). There was no evidence of raised T2 (E: 38 ms) or T1 (1218 ms) values on parametric mapping, and no myocardial late gadolinium enhancement. The cardiac diagnosis adjudicated by the dinical event committee was no evidence of myocardial injury.