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. 2022 Mar 12;38(6):622–636. doi: 10.1007/s12264-022-00826-9

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© Center for Excellence in Brain Science and Intelligence Technology, Chinese Academy of Sciences 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Involvement of CaM and CaMKII in the exendin-4-induced suppression of GABA currents. A, C Representative recordings from two GCs, showing that exendin-4 (50 nmol/L) no longer suppresses the GABA currents during the internal infusion of 100 μmol/L W-7 (A) or extracellular perfusion with 1 μmol/L KN-93 (C). B, D Bar charts summarizing the results for the effects of W-7 (B) and KN-93 (D). n.s., not significant, paired Student’s t-test.