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. Author manuscript; available in PMC: 2022 Jun 18.
Published in final edited form as: Toxicol Lett. 2021 Jan 6;340:33–42. doi: 10.1016/j.toxlet.2021.01.005

Fig. 8.

Fig. 8.

N-acetylcysteine (NAC) treatment to the acrolein exposed hCSFs displayed restoration of the mitochondrial membrane potential as measured by the MitoTracker Red dye (A-D). The quantification data suggested that NAC rescues the mitochondrial dysfunction after acrolein exposure significantly compared to the ACR group (E). Similarly, the caspase 3/7 activity was significantly decreased after NAC treatment in the acrolein exposure (ACR) group (F). All the experiments were independently performed three times using each sample in triplicates. ***p ≤ 0.001 compared to the CTK group, and ###p ≤ 0.001 compared to the ACR group. All the experiments were independently performed three times using each sample in triplicates. The results were presented as mean ± SEM. The one-way analysis of variance (ANOVA) with Bonferroni post hoc test were used for statistical analysis. Scale bar=100 μm.