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. 2022 Jan 28;18(5):1834–1850. doi: 10.1007/s12015-022-10335-x

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Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — Molecular mechanisms through which the hUC-MSC-sec promotes primordial follicle activation. HGF secreted from MSCs promotes the expression of KITL, thereby enhancing the activity of the PI3K-AKT signaling pathway in dormant oocytes and thus activating primordial follicles