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. 2022 Jun 20;19:162. doi: 10.1186/s12974-022-02525-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Analysis of the difference in protein levels of Nav1.6 in ACC induced by SNI and L5-VRT. A Representative western blotting of Nav1.6 protein levels in bilateral ACC following SNI and L5-VRT are shown on the top and the quantification results are shown below. *p < 0.05, **p < 0.01 versus sham group (one-way ANOVA). B The expression of Nav1.6-positive cells in bilateral ACC 7d post-SNI surgery. Representative data obtained from sham and SNI rats are shown. Scale bar = 100 μm. Detection area by red solid rectangular box is shown on the upper right and mean fluorescence intensity is shown on the lower right. ***p < 0.001 versus sham group (one-way ANOVA)