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. 2022 Jun 21;20:291. doi: 10.1186/s12951-022-01504-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Mir-762 is a key mediator of iPSCs-KCs-Exos induced migration of keratinocytes and endothelial cells. A Representative images of the wound healing assay of keratinocytes in each group (left panel). Scale bar = 100 μm. Quantitative analysis of the wound healing rates in each group (right panel). B Images of migrated HUVECs in each group. Scale bar, 200 μm. Quantitative analysis of the migrated cells (right panel). Scale bar, 200 μm. C qRT–PCR analysis of miR-762 expression in HaCaT cells and HUVECs which were treated with iPSCs-KCs-Exos (2 μg/mL) for 24 h. *P < 0.05, **P < 0.01