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. 2022 Jun 8;13:910217. doi: 10.3389/fphar.2022.910217

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Copyright © 2022 Ji, Shi, Li, Hou, Yang, Liu, Duan, Liu, Chen and Peng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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THSWD treatment inhibits the generationthe of ROS, activation of NLRP3 inflammasome and secretion of pro-inflammatory cytokine. (A) Quantification of ROS levels; n = 6 per group. (B–I) The Western blot relative band density of NLRP3, ASC, cleaved-caspase-1, IL-1β, IL-18; n = 6 per group. Each experiment was repeated 3 times. ^** p < 0.01, ^*** p < 0.001 versus sham group, ^# p < 0.05, ^## p < 0.01, ^### p < 0.001 versus MCAO/R group, ^& p < 0.05, ^&& p < 0.01,^&&& p < 0.001 versus MCAO/R + THWSD group.