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. 2022 Jun 8;13:904849. doi: 10.3389/fphar.2022.904849

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Copyright © 2022 Wu, Zhu, Li, Xu, Chang, Du, Cui, Zhang, Zhang and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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CCL attenuates the morphological impairment of bipolar and horizontal cells in the light-exposed retinas. IHC was performed to examine the expression of PKCα (in red) and calbindin D (in red) in the retina. Counterstaining of DAPI (in blue) was performed. The immunopositivity of PKCα and calbindin D in OPL was quantified by ImageJ. (A) Representative micrographs showing the expression of PKCα in the retina. (B) The relative fold change in the immunopositivity of PKCα was plotted against that from Normal control. (C) Representative micrographs showing the expression of calbindin D in the retina. (D) The relative fold change in the immunopositivity of calbindin D was plotted against that from Normal control. INL, inner nuclear layer; OPL, outer plexiform layer. Scale bar, 50 μm. Data were expressed as mean ± SD (n = 6 per group). ^### Compared to Normal control, p < 0.001; *** compared to Light_vehicle, p < 0.001.