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. 2022 Jun 8;12:816504. doi: 10.3389/fonc.2022.816504

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Copyright © 2022 Kalyanaraman, Cheng and Hardy

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Metabolic reprogramming from glycolysis to enhanced OXPHOS occurs in drug-resistant cancer cells. This characteristic makes these cells more vulnerable to mitochondria-targeted OXPHOS inhibitors.