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. 2022 Jun 9;9:885178. doi: 10.3389/fmed.2022.885178

Table 3.

Genotype-phenotype correlations in pediatric patients with steroid-resistant nephrotic syndrome of AD mutation and AR mutation.

AD AR
WT1 TRPC6 ACTN4 Others Total NPHS1 NPHS2 ADCK4 LAMB2 Others Total
(n = 32) (n = 6) (n = 3) (n = 3) (n = 44) (n = 18) (n = 15) (n = 13) (n = 3) (n = 7) (n = 56)
Sex Male:Female 8:24 4:2 1:2 2:1 15:29 14:4 9:6 4:9 0:3 5:2 32:24
Age of onset 1–3 m 3 0 0 0 3 (6.8%) 17 2 1 2 0 22 (39.3%)
3 m−1 y 6 1 0 0 7 (15.9%) 0 3 1 1 1 6 (10.7%)
1–2 y 9 2 0 0 11 (25%) 0 5 3 0 2 10 (17.9%)
3–5 y 5 1 1 1 8 (18.2%) 1 0 2 0 2 5 (8.9%)
6–12 y 7 2 2 2 13 (29.5%) 0 3 6 0 2 11 (19.6%)
≥12 y 2 0 0 0 2 (4.5%) 0 2 0 0 0 2 (3.6%)
Kidney biopsy FSGS 8 4 2 1 15 (34%) 1 6 5 1 5 18 (32.1%)
MCD 1 0 0 0 1 (2.2%) 0 1 0 0 0 1 (1.8%)
MsPGN 1 0 0 0 1 (2.2%) 1 3 1 0 0 5 (8.9%)
Others 3 1 0 1 5 (11.3%) 0 0 0 0 0 0
Not done 19 1 1 1 22 (50%) 16 5 7 2 2 32 (57.1%)
Renal outcome at follow-up Normal eGFR 6 1 0 2 9 (20.5%) 4 8 3 0 3 18 (32.1%)
CKD stages 2–4 4 0 0 0 4 (9.1%) 2 0 0 1 1 4 (7.1%)
ESKD/RRT 9 5 2 1 17 (38.7%) 0 5 6 0 2 13 (23.2%)
Mortality 8 0 1 0 9 (20.5%) 6 0 0 0 1 7 (12.5%)
Data unavailable 5 0 0 0 5 (11.4%) 6 2 4 2 0 14 (25%)
Family history Yes 2 0 0 1 3 (6.7%) 0 5 6 0 1 12 (21.4%)
No 29 6 3 2 40 (91%) 17 10 7 2 5 31 (55.4%)
Data unavailable 1 0 0 0 1 (2.2%) 1 0 0 1 1 3 (5.3%)
Extrarenal manifestations Yes 12 0 0 0 12 (27.2%) 4 4 0 2 3 13 (23.2%)
No 20 6 3 3 32 (72.7%) 14 11 15 1 4 43 (76.8%)
Renal transplantation Yes 2 3 0 0 5 (11.4%) 0 3 2 0 2 9 (16.1%)
No 24 2 3 3 32 (72.7%) 12 8 8 0 2 30 (53.6%)
Data unavailable 6 1 0 0 7 (15.9%) 5 4 3 3 3 17 (30.3%)

AD, autosomal dominant; AR, autosomal recessive. FSGS, focal segmental glomerulosclerosis; MCD, Minimal change disease; MsPGN, Mesangial proliferative glomerulonephritis; eGFR, estimated glomerular filtration rate; CKD, chronic kidney disease; ESKD, end-stage renal disease;RRT, Renal replacement therapy (hemodialysis, peritoneal dialysis, kidney transplantation).