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. 2022 Jun 23;19:43. doi: 10.1186/s12989-022-00482-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Plasma OPN concentrations were correlated with urinary 1-OHP concentration in a human population. Fasting blood samples and urine samples were collected from nonsmoking young adults for analysis. The plot shows plasma OPN concentrations across quartiles of urinary 1-OHP levels. One-way ANOVA and P trend analysis were used to assess the differences and trends in OPN levels. Fisher’s least significance difference (LSD) method was used in post hoc comparisons to test the difference in OPN levels for a two-by-two comparison with the 1-OHP groups