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. 2022 Jun 20;14(12):2553. doi: 10.3390/nu14122553

Figure 3.

Figure 3

Eriodictyol inhibited oxidative stress in BJ cells following H2O2 exposure. (A) Eriodictyol treatment (5–20 µM; 24 h) did not alter the redox status of BJ cells. (B) Eriodictyol pretreatment (10–20 µM; 24 h) inhibited H2O2-induced oxidative stress (500 µM; 1 h) in a concentration-dependent fashion. Oxidative stress was immediately evaluated using the DCFH-DA assay after the indicated treatments. Relative oxidative stress was calculated compared with that in untreated controls. DMSO (0.5%) was used as a vehicle control (n = 3; each biological replicate consisted of three independent wells; mean ± SEM). * p < 0.05 versus untreated controls; p < 0.05 versus H2O2-treated cells. N.S., not significant.