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Potential contributions of innate immunity to the pathogenesis of cardiometabolic HFpEF. Depicted is a working model in which cardiometabolic risk factors, including visceral adiposity and hypertension, fuel immunometabolic mobilization of innate immune cell subsets. This immune cell mobilization, in turn, activates intercellular crosstalk that also regulates myocardial metabolic pathways.
HSPC: hematopoietic stem/progenitor cell
Mϕ: macrophages
IL-1β: interleukin 1β
