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. 2022 Jun 24;17(6):e0269920. doi: 10.1371/journal.pone.0269920

Fig 6. mRNA expression of SGK1 in HRMC in normal and high glucose conditions upon aldosterone and cortisol stimulation.

Fig 6

A) mRNA expression of SKG1 in HRMC. Normal glucose conditions (5.25mM). Baseline control was EtOH. Fold difference is shown with cyclophilin A as endogenous control. Aldosterone (**p = 0.0026) and cortisol (**p = 0.0002) significantly stimulated SGK1 expression in HRMC on a normal glucose background. B) mRNA expression of SGK1 in HRMC. High glucose conditions (25.25mM). Baseline control was EtOH. Fold difference is shown with cyclophilin A as endogenous control. Aldosterone (**p = 0.0018) and cortisol (***p<0.0001) significantly stimulated SGK1 expression in HRMC on a high glucose background. *p<0.05, **p<0.01, ***p<0.0001, ns = not significant.