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. 2022 Jun 24;8:82. doi: 10.1038/s41531-022-00347-2

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Tremor-related activity originates at internal pallidal globus (GPi), which propagate to cortex. Cortex and ventral intermediate nucleus of thalamus (VIM) form a circuit, which is possible the base of tremor-related oscillation. Cerebellum also projects to VIM, this projection possibly modulate amplitude of tremor, while cerebellum was modulated by cerebral cortex. COR cortex, CER cerebellum. Orange arrows indicate projections within cerebello-thalamo-cortical circuit, blue arrow indicates projection from GPi to cortex.