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. 2022 Jun 22;289(1977):20220773. doi: 10.1098/rspb.2022.0773

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© 2022 The Authors.

Published by the Royal Society under the terms of the Creative Commons Attribution License http://creativecommons.org/licenses/by/4.0/, which permits unrestricted use, provided the original author and source are credited.

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Figure 2. — Mutations affecting Buletin cause a specific susceptibility to P. burhodogranaria. (a) Dro^SK3 flies succumb to infection by En. cloacae slightly later than either Dro^SK4 or Dro-AttAB^SK2 flies that lack both Drc and Btn. The ultimate rate of mortality is comparable (p > 0.05 in comparison between these various Dro mutants). (b) Drosocin mutants that retain Btn (Dro^SK3) survive infection by P. burhodogranariea better than flies lacking both Drc and Btn (Dro^SK4, DroAtt^SK2). (c) Wild-type flies with the Threonine allele of the Btn Thr52Ala polymorphism phenocopy the effect of Btn deletion compared to Alanine-encoding iso w¹¹¹⁸ in defence against P. burhodogranariea. OD, optical density. (Online version in colour.)