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. 2022 Jun 27;19:166. doi: 10.1186/s12974-022-02529-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — JNK–STAT3 signaling contributes to HFD-induced astrocytes reactivation. A Graphic illustration of the signaling pathway in astrocytes related to reactivation. B Astrocytes from ND and HFD mice were isolated by MACS and detected by flow cytometry. The astrocytes reactivation related membrane receptors (C) and intracellular modulators (D) were detected by Milliplex assay. E Astrocytes from saline and CNO-injected mice were isolated by MACS and detected by flow cytometry. The astrocytes reactivation related membrane receptors (F) and intracellular modulators (G) were detected by Milliplex assay. (*p < 0.05, ***p < 0.001 compared with ND mice; ^#p < 0.05, ^##p < 0.001 compared with saline-treated mice)