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. 2022 Jun 14;48(2):139. doi: 10.3892/or.2022.8350

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — UBE2T-mediated alleviation of the effects of FANCI knockdown on EMT. (A) Wound-healing and (B) Transwell assays showed that overexpression of UBE2T reversed the FANCI knockdown-induced inhibition of cell invasion and migration. (C) Upregulation of E-cadherin expression and downregulation of N-cadherin and vimentin expression, caused by FANCI knockdown, were reversed when UBE2T was overexpressed. *P<0.05, **P<0.01, ***P<0.001. UBE2T, ubiquitin-conjugating enzyme E2T; FANCI, Fanconi anemia complementation group I; EMT, epithelial-mesenchymal transition; NC, negative control.