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. 2022 Jun 30;23:25. doi: 10.1186/s12860-022-00424-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — CCT3 is overexpressed in lung cancer tissues and cells. A The transcript abundance of CCT3 was analyzed in lung adenocarcinoma (LUAD, n = 483) and normal tissues (n = 347) from TCGA database. *p < 0.05. B The transcript abundance of CCT3 was analyzed in lung squamous cell carcinoma (LUSC, n = 486) and normal tissues (n = 338) from TCGA database. *p < 0.05. C Nine pairs of lung cancer and adjacent normal tissues were subjected to qRT-PCR analysis of CCT3 expression. ***p < 0.001. D Western blot analysis of CCT3 in lung cancer and paired adjacent normal tissues. The blots were cut prior to hybridisation with antibodies during blotting. E and F qRT-PCR (E) and Western blot (F) analysis of CCT3 in normal lung cells Beas-2B and in cancer cells H2228, H1299 and H1975. **p < 0.01. GAPDH is the internal control. The blots were cut prior to hybridisation with antibodies during blotting