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. 2022 Jun 30;23:25. doi: 10.1186/s12860-022-00424-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig.6 — CCT3 stimulates AKT activity. A Ctrl and CCT3 overexpressed H1975 cells, shCtrl, shCCT3#1 and shCCT3#2 H2228 cells were subjected Western blot analysis with indicated antibodies. The blots were cut prior to hybridisation with antibodies during blotting. B Cell viability was the determined by CCK8 assay in Ctrl, CCT3 overexpressed, Ctrl and CCT3 overexpressed H1975 cells that were treated with MK2206 (2 uM) for indicated time. *p < 0.05. **p < 0.01. C Schematic diagram of how CCT3 contributes to LUAD cell growth.