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. 2022 Jun 30;12:11041. doi: 10.1038/s41598-022-15227-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Immunofluorescence analysis of mice sections vimentin (panels a–d); col1A (panels e–h); FAP1α (panels i–I). Densitometry analysis of fluorescence intensity of the same protein markers (B) for vimentin; (C) for col1A; (D) for FAP1α). The data represent a mean of n = 3 independent experiments ± SD based on one-way ANOVA and a two-tailed Student's t-test as appropriated, *p < 0.05, **p < 0.01, ***p < 0.001 versus untreated control; ^#p < 0.05, ^##p < 0.01, ^###p < 0.001 for combination versus mesoglycan or VEGF treated cells; ¶¶p < 0.01; ¶¶¶p < 0.001 for VEGF treatment versus mesoglycan one.