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. 2022 Jun 17;13:892097. doi: 10.3389/fphar.2022.892097

TABLE 2.

[35S]-GTPγS binding potencies for human S1P1–S1P5.

Compound Human S1P1 Human S1P2 Human S1P3 Human S1P4 Human S1P5
EC50 (nM) IA (%) EC50 (nM) IA (%) EC50 (nM) IA (%) EC50 (nM) IA (%) EC50 (nM) IA (%)
Ozanimod 0.4 ± 0.03 85 ± 1 >10000 34.8 ± 1 >1111 89.4 ± 7.4 1486.6 ± 306.5 39.4 ± 5.3 5.84 ± 0.51 97 ± 3.7
FTY720-p 0.2 ± 0.01 84.9 ± 2.3 >1000 33.2 ± 1 1.33 ± 0.19 106.5 ± 8.3 2.06 ± 0.3 59.2 ± 5.4 0.49 ± 0.07 74 ± 5.1
Amiselimod-p 0.15 ± 0.01 84.1 ± 1.6 >4000 NR 18.98 ± 2.98 29.1 ± 1.6 2.23 ± 0.13 122.2 ± 3.6 0.58 ± 0.08 76.6 ± 6.9
Ponesimod 3.42 ± 1.17 85.8 ± 6.2 >10000 NR 89.52 ± 14.29 82.1 ± 7.5 >10000 26.5 ± 2.5 43.18 ± 11.99 92.9 ± 9.8
Siponimod 0.46 ± 0.05 82.1 ± 0.9 >10000 NR >1111 90.7 ± 2.5 383.73 ± 67.82 81.5 ± 2.4 0.3 ± 0.02 102 ± 9.1
KRP-203-p 0.26 ± 0 85.7 ± 3.6 >10000 NR 3.17 ± 0.26 25.7 ± 3.8 3.13 ± 0.29 56.3 ± 3.8 0.69 ± 0.19 45.8 ± 7.6
Etrasimod 5.48 ± 0.46 86.6 ± 1.1 >10000 NR 1164.3 ± 354.1 81.2 ± 17 1125.2 ± 188.9 40.4 ± 4.6 58.87 ± 6.75 82 ± 4.4

[35S]-GTPγS binding in response to increasing concentrations of test compound performed using Chinese hamster ovary cell membranes stably expressing recombinant human S1P1–S1P5. Data shown are the mean ± standard error of the mean of the compound potencies as determined by the concentration required to elicit a half-maximal response (EC50) as well as the intrinsic activity (IA) relative to the maximal response generated with the endogenous ligand, S1P, which was taken to be 100%, calculated for 3–5 replicate experiments run in duplicate wells. NR indicates no response where the maximal response was less than 10% of that of S1P. Italic type indicates that the response was achieved at the top test compound concentration of 10000 nM, where no clear maximal response was defined.

[35S]-GTPγS, [35S]-guanosine-5'-(γ-thio)-triphosphate; S1P, sphingosine 1-phosphate; S1P1–S1P5, sphingosine 1-phosphate receptor subtypes 1 through 5.