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. 2022 Jun 17;24(2):267. doi: 10.3892/ol.2022.13387

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — LPA reduces oxidative stress injury of DOX-induced HeLa cells. (A) Reactive oxygen species fluorescence intensity levels of the control, LPA, DOX and LPA + DOX HeLa cell groups. (B) SOD levels of the control, LPA, DOX and LPA + DOX HeLa cell groups. (C) MDA levels of the control, LPA, DOX and LPA + DOX HeLa cell groups. Data are presented as the mean ± SD (n=3). *P<0.05 and **P<0.01 vs. the control. DOX, doxorubicin hydrochloride; LPA, lysophosphatidic acid; SOD, superoxide dismutase; MDA, malondialdehyde; n.s., no significance.