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. 2022 Jul 4;22:729. doi: 10.1186/s12885-022-09791-y

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Variation of m7G related genes in glioblastoma. A CNV of m7G related genes in somatic cells. The mutation frequency is listed on the right. B Copy number of each m7G related gene in detail. GAIN infers to amplification and LOSS indicate deletion. C Gene location on chromosome with mutation information. Blue dots are identical to deletion and red dots are amplification. D Expression level of m7G related genes in normal and Glioblastoma samples. E Expression modification of m7G related genes and their roles in regulation. F Cumulative distribution function curve proves the most effect way of clustering. G Grouping based on the expression of m7G related genes. Group 1 indicate group A and group 2 means group B. H Group A and B are separated, proving the significance of grouping. I Survival analysis between group A and B. P < 0.05 is witnessed as significant