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. 2022 Jun 30;11(13):2078. doi: 10.3390/cells11132078

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Integrin β3 and AXL were significantly upregulated in erlotinib-resistant (HCC827 ER and HCC4006 ER) cell lines compared with parental (HCC827 and HCC4006) NSCLC cell lines. (A,B) The mRNA levels of integrin β3 and AXL were determined by qRT-PCR in erlotinib-resistant cells compared with parental cells. GAPDH was used as a loading control. (C,D) Western blot assays showing integrin β3 and AXL expression in erlotinib-resistant and parental cells. β-actin was used as a loading control. n = 3 independent experiments.