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. 2022 Jun 27;13:915244. doi: 10.3389/fimmu.2022.915244

Table 1.

Primers used for the response to infection.

Gene Forward primer (5’-3’) Reverse primer (5’-3’)
ACTB GGAAATCGTGCGTGACATTA AGGAGGAAGGCTGGAAGAG
GAPDH Hs02786624_g1
M1 genes
TNF AGGAGAAGAGGCTGAGGAACAAG GAGGGAGAGAAGCAACTACAGACC
IL1B CAGCACCTCTCAAGCAGAAAAC GTTGGGCATTGGTGTAGACAAC
IL6 CCAGGAGAAGATTCCAAAGATG GGAAGGTTCAGGTTGTTTTCTG
IFNG GTTTTGGGTTCTCTTGGCTGTTA ACACTCTTTTGGATGCTCTGGTC
CXCL10 TCCCATCTTCCAAGGGTACTAA GGTAGCCACTGAAAGAATTTGG
M2 genes
IL10 GGGGGTTGAGGTATCAGAGGTAA GCTCCAAGAGAAAGGCATCTACA
TGFB GACATCAAAAGATAACCACTC TCTATGACAAGTTCAAGCAGA
IL1RA TCTATCACCAGACTTGACACA CCTAATCACTCTCCTCCTCTTCC
CD163 CGGTCTCTGTGATTTGTAACCAG TACTATGCTTTCCCCATCCATC
BTN isoform genes
BTN3A1 TTCCAGGTCATAGTGTCTGC TGAGCAGCTGAGCAAAAGG
BTN3A2 TGGGAATACCAAGGGA AGTGAGCAGCTGGACCAAGA
BTN3A3 GAGGGAATACTAAGAAATGGT GAAGAGGGAGACATGAAAGT
BTN2A1 Hs00924832_m1
BTN2A2 Hs00950165_g1
C. burnetii gene
CB COM-1 GCACTATTTTTAGCCG-GAACCTT TTGAGGAGAAAAACTGGATTGAGA
MTB ITS CAAGGCATCCACCATGCGC GGGTGGGGTGTGGTGTTTGA