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. 2022 Jul 11;6(3):e194–e212. doi: 10.1055/a-1801-2055

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution License, permitting unrestricted use, distribution, and reproduction so long as the original work is properly cited. ( https://creativecommons.org/licenses/by/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 1 — Schematic presentation of structural domains of membrane thrombomodulin (TM) ( A ) and TM mutations ( B ), its release mechanisms of predicted sTMs with corresponding domains (C), and microvesicle-TM ( C ). CS, chondroitin sulfate; CTLD, C-type lectin-like domain; EGF, epidermal growth factor; RHBDL2, the intramembrane protease rhomboid-like-2; Ser, serine; sTM, soluble thrombomodulin; Thr, threonine.