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. 2022 Jun 2;13(5):13654–13666. doi: 10.1080/21655979.2022.2084246

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — LINC00514 expression led to NSCLC cancer progression by modulating the Wnt/β-Catenin signaling. (a) Colony formation assay showed that silencing LINC00514 resulted in decreased cell proliferation, restored by the Wnt agonist LiCl. (b) Transwell assay further showed that LiCl could recover migration and invasion rate of LINC00514 knockdown H1299/A549 cells. LiCl, Lithium chloride; Data are presented as the mean ± SD. **p < 0.01, compared with sh-NC group; ##p < 0.01, compared with sh-LINC00514 group, N = 3.