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. 2022 Jun 27;23(7):1076–1085. doi: 10.1038/s41590-022-01230-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 2 — a, Pie chart shows the number of antigen-specific MBC sequences (in parenthesis, clonal families) from D1 identified through the isolation of monoclonal antibodies against influenza virus (FLU), measles virus (MV), respiratory syncytial virus (RSV), and tetanus toxoid (TT). b, Genealogical trees show representative recall antigen-specific clonal families in D1. Isotypes are color-coded (x=isotype not determined). Somatic mutations at nucleotide and at amino acid (in parenthesis) levels is indicated on individual branches of the trees. The vertical thin dotted lines without arrowheads connect cells with identical VH/VL sequence. The thickness of the arrows reflects the number of mutations. More examples can be found in the RepSeq Playground.