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. 2022 Jul 11;13(7):597. doi: 10.1038/s41419-022-05040-z

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A SNHG25 expression was analyzed in NBL tissues and normal neural tissues from the combined cohort of TCGA TARGET GTEx in UCSC-XENA. B SNHG25 expression in NB cell lines, MCF-10A, and HEK-293T cells were detected by RT-qPCR. (One-way ANOVA, Tukey) C The interference efficiency of sh-SNHG25 was detected in SK-N-AS and SK-N-SH cells by RT-qPCR. (One-way ANOVA, Dunnett) D–F CCK-8 assay, colony formation assay, and flow cytometry analysis detected proliferation and apoptosis of NB cells with SNHG25 silence. (One-way ANOVA, Dunnett) G–I Transwell and wound healing assays detected invasion and migration of NB cells with SNHG25 silence. (One-way ANOVA, Dunnett) ^**P < 0.01. Error bars indicate mean ± SD. (N = 3).