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. 2022 Jul 13;13:4071. doi: 10.1038/s41467-022-31742-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — BDF5 localised to chromatin with the CRKT complex members depicted as interacting directly and influencing transcription, either directly or through a putative PAF1-like complex. The red triangle indicates a disruption of transcription once BDF5 is deleted. Other proximal processes are indicated in grey boxes. Indications of detected acetylation and phosphorylation are depicted as yellow or blue dots respectively. Juxtaposition and stoichiometry of complex members are for illustration only, not to scale.