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. 2022 Jul 5;66:10.29219/fnr.v66.5459. doi: 10.29219/fnr.v66.5459

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© 2022 Yiying Chen et al.

This is an Open Access article distributed under the terms of the Creative Commons Attribution 4.0 International License, allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material for any purpose, even commercially, provided the original work is properly cited and states its license.

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Fig. 5 — Effect of L. plantarum Lp2 on hepatic NFκB signaling. TLR4, p-NFκB, NFκB, I-κB, and Caspase-3 protein expressions in liver. β-actin was used as a control for the protein blots (A). Relative protein levels of TLR4(B). The levels of the phosphorylation of NFκB p65 were increased in the LPS group, Lp2 inhibited the LPS-induced increase of the phosphorylation of NFκB p65 (C). Values are expressed as mean ± SEM. *P < 0.05 and **P < 0.01 compared with the LPS group.