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. 2022 Jul 16;13(7):617. doi: 10.1038/s41419-022-05078-z

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A Differential gene expression analysis identified 19 overlapping genes that were significantly higher expressed in PDOs expressing KIT compared to those without KIT expression. B Scatter plots showing the correlation of the identified KIT-dependency gene signature with CMS1-4-identifying genes in the CMS-3232 CRC cohort [1]. Bar plot showing Pearson R values of (C). D Score for the KIT-dependency gene signature per CMS-annotated tumor in the CMS-3232 cohort.