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. 2022 Apr 20;30(4):1431–1443. doi: 10.1007/s10787-022-00967-3

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Basal NQO1 activity, NQO1, HMOX1, SOD1 and TXNRD1 mRNA expression in alveolar macrophages from S and COPD patients. Alveolar macrophages were isolated from S and COPD patients. Basal levels of NQO1 activity were assessed by NQO1 activity enzyme assay (A) (n = 8 S and n = 8 COPD). Basal mRNA expression of NQO1 (n = 8 S and n = 8 COPD), HMOX1 (n = 8 S and n = 8 COPD), SOD1 (n = 6 S and n = 8 COPD) and TXNRD1 (n = 6 S and n = 8 COPD) was assessed by RT-qPCR relative to endogenous control (2^−ΔC_t) (B–E respectively). Data represent individual patients with mean. * = significant difference between groups (p < 0.05)